COVID-19 accelerates the progression of atherosclerotic plaques, increasing coronary inflammation and the risk of high-risk ...

COVID-19 isn't just a respiratory illness — it can silently attack the heart. New research reveals that the virus accelerates ...

Lipid-rich, noncalcified coronary plaques are considered more likely to rupture and are associated with a higher risk of potentially fatal ischemic cardiac events than fibrous or calcified lesions.

The virus that causes COVID-19 may expedite progression of coronary lesions to high-risk plaque. SARS-CoV-2 was linked to significantly elevated risk for cardiac death, MI and revascularization.

A new study found severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection was associated with the rapid growth ...

Following publication of our studies, some physicians and patients question whether coronary atherosclerosis in athletes requires statins due to the potential lower non-calcified plaque burden and ...

Lesions in patients with SARS-CoV-2 infection had a higher incidence of developing into high-risk plaques (20.1% versus 15.8%) and coronary inflammation (27% versus 19.9%).